ASSIGNMENT HELP | Discuss the pathogen and it’s proliferation.

A 22 year old presents with vaginal D/C, characteristic of green, frothy, odorous upon exam. The diagnosis is Trichimonas Vaginosis.

Discuss the pathogen and it’s proliferation.
Discuss male partner treatment. How does metronidazole target the urethral canal?
Co-infections are common with STIs. What is the pathogenesis of Chlamydia Infectgion?


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Sexually Transmitted Disease Case Study

Trichomoniasis vaginosis is a sexually transmitted infection caused by a single-celled parasite commonly referred to as Trichomonas vaginalis (Tv). This parasite is an extracellular pathogen that facilitates adherence to the epithelial cells in order to inhabit the infected human. Additionally, the parasite has been known to interface with the immune system of a person including the vaginal | PLACE YOUR ORDER NOW AT | modes of this pathogens comprises of damage to the host tissue that occurs when the parasites kills the cells thus upsetting the stable state of the vaginal microbial ecology and further eliciting inflammation through the activation of the human’s immune | PLACE YOUR ORDER NOW AT | parasite commonly infects the urethra, vulva, vagina and the penis.

While the male infection of Trichomonas vaginalis is generally asymptomatic, some may experience relatively mild symptoms. Just like women, the condiction can be cured through the use of metronidazole antibiotic twice a day for a period of 5 to 7 | PLACE YOUR ORDER NOW AT |

Metronidazole acts by entering the T. vaginalis through passive diffusion. The medication itself is naturally sedentary but its anaerobic reduction causes creation of a cytotoxic nitro radical anion | PLACE YOUR ORDER NOW AT | radical gets conjectured in order to bind | PLACE YOUR ORDER NOW AT | to the host’s DNA, thus upsetting the strands of its cells leading to expiry of the Tv cells.

The pathogenesis of Chlamydia Infection occurs when the host reaction to chlamydiae is instigated by epithelial cells that are the key targets of infection (Elwell, Mirrashidi & Engel, 2016). The | PLACE YOUR ORDER NOW AT |  host epithelial cells act as the first responders where they start and spread immune responses.


Mercer, F., & Johnson, P. J. (2018). Trichomonas vaginalis: Pathogenesis, symbiont interactions, and host cell immune responses. Trends in Parasitology34(8), 683-693.

de Aquino, M. F. K., Hinderfeld, A. S., & Simoes-Barbosa, A. (2020). Trichomonas vaginalis. Trends in Parasitology36(7), 646-647.

Edwards, T., Burke, P., Smalley, H., & Hobbs, G. (2016). Trichomonas vaginalis: Clinical relevance, pathogenicity and diagnosis. Critical Reviews in Microbiology42(3), 406-417.s

Elwell, C., Mirrashidi, K., & Engel, J. (2016). Chlamydia cell biology and pathogenesis. Nature Reviews Microbiology14(6), 385-400.


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